An interdisciplinary and multinational crew of experts current contributions describing the present prestige of vaccines opposed to virally brought about tumors and speak about the capacity in which they are often more suitable.
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Extra resources for Ciba Foundation Symposium 187 - Vaccines Against Virally Induced Cancers
Acknowledgements N. Coleman was supported by a Clinical Training Fellowship from the Imperial Cancer Research Fund and Mark Chambers by the Nita King Studentship (Department of Pathology University of Cambridge), The Ts-iao Ch-hia Research Studentship (Downing College University of Cambridge) and the Harnett Fund (Clinical School University of Cambridge). References Chambers M, Zhang W , Coleman N, Nash AA, Stanley MA 1994a Modulation of the DTH response to HPV 16 E7. In: Stanley M (ed) Immunology of human papillomaviruses (HPV’s).
J Gen Virol 71:1355-1364 Ford JN, Jennings PA, Spradbrow PB, Francis J 1982 Evidence for papillomaviruses in ocular lesions in cattle. Res Vet Sci 32:257-259 Hoffmann D, Jennings PA, Spradbrow PB 1981 Immunotherapy of bovine ocular squamous cell carcinomas with phenol-saline extracts of allogenic carcinomas. Aust Vet J 57:159-162 Kawakami Y, Eliyahu S, Delgado CH et al 1994 Cloning of the gene encoding for a shared human melanoma antigen recognized by autologous T-cells infiltrating into tumor.
The warts regress and you don’t see them again unless you are immunosuppressed. The other extreme is HPV-16, where the immune response is much weaker and the virus is much more difficult to get rid of (Tindle & Frazer 1994). Whether that’s a property of the viral proteins or of the nature of the viral life cycle is still up for grabs. Cheever: Are there some antigens to which you do get a vigorous response and some, such as the transforming antigens, to which you don’t? Is there a vigorous immune response in patients with regressing lesions?